Study 86 Quiz # 5 (Vitamins and Minerals) flashcards from Samantha V. on StudyBlue. Describe the relationship between folate and vitamin B Folate. Background. Historic reports on the treatment of pernicious anemia with folic acid suggest that high-level folic acid fortification delays the diagnosis of or. B Water-Soluble Vitamin Deficiency & Toxicity Symptoms to know to pass the quiz include the definition of terms used in relation to vitamin B12 deficiency.
Abstract Background Historic reports on the treatment of pernicious anemia with folic acid suggest that high-level folic acid fortification delays the diagnosis of or exacerbates the effects of vitamin B deficiency, which affects many seniors.
This idea is controversial, however, because observational data are few and inconclusive.
Furthermore, experimental investigation is unethical. Results After control for demographic characteristics, cancer, smoking, alcohol intake, serum ferritin, and serum creatinine, low versus normal vitamin B status was associated with anemia [odds ratio OR: Conclusion In seniors with low vitamin B status, high serum folate was associated with anemia and cognitive impairment.
When vitamin B status was normal, however, high serum folate was associated with protection against cognitive impairment. The main goal of this program is the prevention of neural tube birth defects 1. The elderly are of particular concern because of age-related declines in vitamin absorption and extraction of vitamin B from protein 4 — 11 and age-related increases in autoimmunity against intrinsic factor or the gastric parietal cells that produce it 11 — Fears of harm from fortification to seniors and other Americans with low vitamin B status are based on early case reports of pernicious anemia that detail the alleviation of anemia but the precipitation or exacerbation of neurologic or neuropsychiatric sequelae after folic acid administration—an early form of treatment based on the mistaken idea that a lack of folate was the problem 8 Modern reports on the effects of high folic acid intakes are infrequent because of the rarity of identified cases of vitamin B deficiency affected by oversupplementation or mistreatment with folic acid.
However, close study of the original case reports showed no proof that folic acid therapy exacerbated central nervous system CNS —related symptoms 1516and modern case reports and studies suggest that folic acid therapy did not cure anemia either 17 — Such data support the idea that folic acid can be safely added to foods in moderation, but recent editorials have stressed the need for systematic study of the hypothesis that high folate intakes cause harm 21 Data collected in the most recent National Health and Nutrition Examination Survey NHANES afforded us the opportunity to study interrelations between serum folate and vitamin B status in relation to anemia, macrocytosis, and cognitive impairment in the age of folic acid fortification.
The survey is currently implemented as a continuous annual survey and uses a complex multistage probability design to select a representative sample of the noninstitutionalized US civilian population. To increase the precision of estimates derived from the survey, adolescents, the elderly, Mexican Americans, and blacks are oversampled.
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Consistent with NHANES analytic guidelines, we combined data from the 2 most recent surveys into a single data set — Trained interviewers used a computer-assisted personal interview system to interview participants in their homes.
J Am Geriatr Soc. Cognitive recovery with vitamin B12 therapy: A longitudinal neuropsychological assessment. Am J Med Sci. Folate status, vascular disease and cognition in elderly Canadians. Low folate levels in the cognitive decline of elderly patients and efficacy of folate as a treatment for improving memory deficits.
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Nutritional status and cognitive functioning in a normally aging sample: Am J Clin Nutr. Homocysteine is a risk factor for cerebral small vessel disease, acting via endothelial dysfunction. Meta-analysis of the Hachinski Ischemic Score in pathologically verified dementias.What Is webob.info simple terms!
Brain infarction and the clinical expression of Alzheimer disease. Cognitive performance and plasma levels of homocysteine, vitamin B12, folate and lipids in patients with Alzheimer disease.
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Homocysteine, folate, and vitamin B in mild cognitive impairment, Alzheimer disease, and vascular dementia. Correlations between cognitive, behavioural and psychological findings and levels of vitamin B12 and folate in patients with dementia. The effect of vitamin B12 deficiency on older veterans and its relationship to health. Folate and cobalamin in psychiatric illness. Age related changes in cobalamin vitamin B12 handling.
B12: Water-Soluble Vitamin Deficiency & Toxicity Symptoms
Neurotoxicity associated with dual actions of homocysteine at the N-methyl-D-aspartate receptor. Low thyroid stimulating hormone in cases with dementia. Low thyroid-stimulating hormone as an independent risk factor for Alzheimer disease.